90 Tuesday June 27, 2000: Workshop Abstracts W:I5 Genetics of Risk Factors for CVD we are measuring levels of gene products involved in atherogenesis in the arterial wall. For example, we have found strong evidence for genetic control of serum levels of soluble P-se&tin (h’ = 0.70), and have detected major QTLs on chromosome 15 (LOD = 3.8) and chromosome 12 (LOD = 2.6). Conclusions: Genomic searches in families are a powerful strategy to identify new QTLs that influence CVD risk factors. I TuW2.15 Genetic ...
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